Welcome to the world of sugar free joy!
Low Carb Sugar-Free Sweets & Cakes
Artinci was born out of Aarti's and Sumit's (Artinci's founders) abiding love for great-tasting dessert, while helping them stay committed to their health goals as well. As a result, Artinci makes delicious desserts with zero sugar, that are science and evidence-backed.
Aarti and Sumit come from a family of three generations of diabetics. They were themselves diagnosed pre-diabetic in 2012, and right there began a lifelong quest of a healthy, active lifestyle, including healthy swaps in food
Sugar free Sweets & Cakes
Sugar Free Kaju Katli (Stevia Sweetened) | Keto, Vegan & Diabetic Friendly Sweet | No Maltitol
Vanilla & Chocolate Marble Sugar free Cake - Diabetic-Friendly, Keto, Gluten-Free (contains egg)
Aarti Laxman (Founder)
Artinci is founded by Aarti Laxman, a certified Metabolic coach in the Low-Carb Nutrition & Metabolic Health domain from dLife.in, India’s only legally tenable course in this subject—recognized by the NSDC (under the Ministry of Skill Development & Entrepreneurship, Govt. of India). It’s also internationally accredited by the CPD Standards Office UK, with a global record of 144 CPD hours—the highest for any course of its kind. The accreditation is both nationally valid and globally recognised in over 50+ countries..
Festive Gifting in Artinci
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All about Sugar and sugar-free
हां, व्यायाम चीनी के एपिजेनेटिक उम्र बढ़ाने वाले प्रभावों को काफी हद तक कम कर सकता है, जैसा कि कई अध्ययनों से पता चलता है। नियमित एरोबिक और स्ट्रेंथ ट्रेनिंग एपिजेनेटिक क्लॉक को धीमा करती है, भले ही चीनी का सेवन हो। डायबिटीज प्रभावित भारतीयों के लिए योग और वॉकिंग आदर्श हैं। एपिजेनेटिक उम्र बढ़ना और चीनी का प्रभाव चीनी डीएनए मिथाइलेशन को बदलकर एपिजेनेटिक उम्र बढ़ाती है, जिससे कोशिकाएं जैविक रूप से पुरानी हो जाती हैं। प्रत्येक ग्राम अतिरिक्त चीनी 0.02 वर्ष एपिजेनेटिक उम्र बढ़ाती है। व्यायाम इन परिवर्तनों को उलट सकता है। व्यायाम कैसे मुकाबला करता है संरचित व्यायाम (जैसे HIIT या स्ट्रेंथ) मसल टिश्यू में एपिजेनेटिक परिवर्तनों को कम करता है। आठ सप्ताह की ट्रेनिंग से महिलाओं की एपिजेनेटिक उम्र 2 वर्ष घटी। यह इम्यूनोसेंसेंस और हृदय जोखिम को कम करता है। अध्ययनों के प्रमाण Aging-US अध्ययन में व्यायाम एपिजेनेटिक एजिंग को धीमा करता है, हृदय, लीवर और गट में लाभ। चूहों में एंड्योरेंस ट्रेनिंग आयु-संबंधी बदलाव उलटती है। व्यायाम चीनी जलाकर उसके प्रभाव को न्यूट्रलाइज करता है। भारतीय संदर्भ में व्यायाम सुझाव डायबिटीज वाले लिए 30 मिनट दैनिक वॉकिंग या योग चीनी प्रभाव कम करता है। सुबह व्यायाम ब्लड शुगर स्थिर रखता है। कम प्रभाव वाले जैसे सूर्य नमस्कार एपिजेनेटिक लाभ देते हैं। https://www.aging-us.com/article/206278/pdf https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2821611 https://pmc.ncbi.nlm.nih.gov/articles/PMC5514203/ https://www.frontiersin.org/journals/genetics/articles/10.3389/fgene.2021.652497/full
How Much Added Sugar Accelerates Epigenetic Age Per Gram
Added sugar accelerates epigenetic aging at a rate of 0.02 years per gram, according to a key JAMA study on midlife women. This means common intakes like 50g daily (two sodas) add nearly a year to your biological age. For diabetes-focused audiences, cutting below 25g daily preserves youthful epigenetics. The Science: 0.02 Years Per Gram In a study of 342 Black and White women, each gram of added sugar raised GrimAge2 epigenetic age by β=0.02 years (95% CI: 0.01-0.04). GrimAge2 measures mortality-linked DNA methylation changes. This holds independently of overall diet quality. Daily Intake Risks Average U.S. intake (77g) accelerates aging by 1.5 years biologically. Indian diets often exceed via sweets; 25g WHO limit halves this effect. Exceeding 35g correlates with faster cellular senescence. Factors Amplifying Acceleration Inflammation from sugar worsens epigenetics in diabetes-prone groups. Midlife women see strongest links; Black women face higher baseline risks. Pair with low Mediterranean diet scores for compounded aging. Cut Sugar to Reverse Trends Dropping added sugar slows GrimAge2; healthy diets reverse up to 2.5 years biologically. Track via apps; prioritize whole foods over processed. https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2821611 https://pubmed.ncbi.nlm.nih.gov/39073813/ https://www.ucsf.edu/news/2024/07/428121/healthy-diet-less-sugar-linked-younger-biological-age https://www.healthline.com/health-news/healthy-diet-less-sugar-biological-aging
Foods That Replace White Sugar to Slow Epigenetic Aging
White sugar speeds up epigenetic aging by altering DNA methylation patterns, making cells biologically older than their chronological age. Replacing it with nutrient-dense alternatives supports healthier epigenetic clocks, as shown in studies on midlife women. These swaps align with diabetes-friendly Indian diets you often cover. Why White Sugar Accelerates Epigenetic Aging Added sugars like white sugar raise epigenetic age independently of overall diet quality. Each extra gram consumed links to older cellular profiles via GrimAge2 clocks. Antioxidant-rich, low-sugar foods counteract this by reducing inflammation and oxidative stress. Top Natural Replacements These alternatives provide sweetness without spiking blood sugar or harming epigenetics. Food Alternative Key Benefits for Epigenetic Aging Indian Diet Fit Glycemic Index Monk Fruit Zero-calorie; antioxidants protect DNA methylation Tea, kheer 0 Stevia Plant-based; anti-inflammatory compounds slow aging clocks Curd, subzi 0 Erythritol Minimal metabolic impact; supports cellular repair Halwa, laddoo 1 Dates (2-3 daily) Fiber + polyphenols combat sugar-induced aging Chutney, smoothie 42 Jaggery (limited) Minerals aid epigenetics over refined sugar Gur chai 65 How to Use in Daily Meals Start breakfast with stevia-sweetened oats or monk fruit yogurt. Use erythritol in low-glycemic desserts like besan barfi for diabetes control. Pair dates with nuts for mid-meal snacks to stabilize blood sugar and epigenetics. Science Behind the Slowdown Healthy diets high in anti-inflammatory nutrients (e.g., from berries or greens replacing sugar) yield younger epigenetic ages. Studies confirm low added sugar under 25g/day preserves telomere length and GrimAge2 markers. This ties into gut health via microbiome support from fibers in dates. https://jamanetwork.com/journals/jamanetworkopen/fullarticle/2821611 https://pubmed.ncbi.nlm.nih.gov/39073813/ https://www.nutritional-psychology.org/women-who-eat-healthier-and-reduce-sugar-intake-age-more-slowly/
Mechanisms by Which Sugar Raises Blood Pressure and Lipids
Sugar, particularly added sugars like fructose, disrupts metabolic pathways to elevate blood pressure and harmful lipids, accelerating cardiovascular risks. This blog breaks down the key biochemical processes, supported by recent research, for health-conscious readers focused on diabetes and nutrition. Fructose and Uric Acid Production Fructose metabolism in the liver rapidly depletes ATP via fructokinase, leading to AMP breakdown and uric acid surge.Elevated uric acid activates NADPH oxidase, causing mitochondrial oxidative stress and reduced energy production.This triggers vasoconstriction and sodium retention, directly raising blood pressure while promoting liver fat synthesis. Insulin Resistance and Sympathetic Activation High sugar intake induces hyperinsulinemia, stimulating the sympathetic nervous system (SNS) and increasing noradrenaline.SNS overdrive boosts renin-angiotensin-aldosterone system (RAAS), enhancing sodium reabsorption and fluid volume for hypertension.Simultaneously, insulin resistance impairs lipid clearance, elevating triglycerides and VLDL. Sodium Handling and Osmolality Effects Sugar enhances gut sodium absorption and renal reabsorption via Na-H exchanger and sodium-chloride cotransporter.High osmolality from salt-sugar combos amplifies endogenous fructose production through the polyol pathway (glucose to sorbitol to fructose).This vicious cycle worsens endothelial dysfunction and vascular stiffness, compounding blood pressure rises. Lipid Dysregulation Pathways Fructose promotes de novo lipogenesis in the liver, converting excess carbs to triglycerides and LDL precursors.Hyperinsulinemia suppresses fat oxidation while increasing free fatty acids, fueling atherogenic dyslipidemia.Result: Higher apoB-containing particles that deposit in arteries, independent of total calories. https://academic.oup.com/ckj/article/16/8/1239/7085008 https://pmc.ncbi.nlm.nih.gov/articles/PMC4336865/ Vasopressin and Leptin Surge Fructose-induced ATP drops elevate vasopressin, causing vasoconstriction via V1a receptors and aldosterone via V1b.Leptin rises in response, further activating SNS and RAAS for sustained hypertension.These hormones link sugar to obesity-independent metabolic syndrome features.

